By E.J. McShane (Eds.)

Hormones and Signaling specializes in the mechanisms of gene law on the mobile point. It additionally describes the activities of hormones in modulating gene rules and animal improvement. Key beneficial properties* Glococorticoid and mineralcorticoid signaling* Orphan nuclear receptors* Nuclear receptor coactivators* Cytokines and STAT signaling* Coordination of cAMP signaling occasions via PKA anchoring* G protein-coupled extracellular Ca2+ (Ca2+0)-sensing receptor (CaR)* Pancreatic islet improvement* Genetic research of androgen receptors in improvement and illness* Antioprogestin regulable gene change for induction of gene expression in vivo* Steroid receptor knockout version

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Hormones and Signaling

Hormones and Signaling makes a speciality of the mechanisms of gene legislation on the mobile point. It additionally describes the activities of hormones in modulating gene legislation and animal improvement. Key positive aspects* Glococorticoid and mineralcorticoid signaling* Orphan nuclear receptors* Nuclear receptor coactivators* Cytokines and STAT signaling* Coordination of cAMP signaling occasions via PKA anchoring* G protein-coupled extracellular Ca2+ (Ca2+0)-sensing receptor (CaR)* Pancreatic islet improvement* Genetic research of androgen receptors in improvement and sickness* Antioprogestin regulable gene change for induction of gene expression in vivo* Steroid receptor knockout version

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And Schu¨tz, G. (1998). DNA binding of the glucocorticoid receptor is not essential for survival. Cell 93, 531–541. , Logan, S. , and Garabedian, M. J. (1998). Antagonism of glucocorticoid rceptor transcriptional activation by the c-Jun N-terminal kinase. Proc. Natl. Acad. Sci. USA 95, 2050–2055. Ross, M. , Evinger, M. , Hyman, S. , Carroll, J. , Reis, D. , Joh, T. , and Goodman, H. M. (1990). Identification of a functional glucocorticoid response element in the phenylethanolamine N-methyltransferase promoter using fusion genes introduced into chromaffin cells in primary culture.

Endocrinol. 7, 528–542. Chang, S. , Mathew, P. , Shimkets, R. , Rossier, B. , and Lifton, R. P. (1996). Mutations in subunits of the epithelial sodium channel cause salt wasting with hyperkalaemic acidosis, pseudohypoaldosteronism type 1. Nature Genetics 12, 248–253. Chapman, M. , Askew, D. , and Miesfeld, R. L. (1996). Transcriptional control of steroid-regulated apoptosis in murine thymoma cells. Mol. Endocrinol. 10, 967–978. Cole, T. , Blendy, J. , Monaghan, A. , and Schu¨tz, G. (1995). Targeted disruption of the glucocorticoid receptor gene blocks adrenergic chromaffin cell development and severely retards lung maturation.

1993). In vitro, submicromolar concentrations of oleoyl-CoA displace triiodothyronine (T3) bound to T3R. Although no studies were performed to determine if these agents altered T3R activity, acyl-CoAs might play a physiologic role in modulating thyroid-hormone-induced lipogenesis by antagonizing T3 binding to its receptor. , 1998). 6 ȐM ): other long-chain acyl-CoAs also bound to HNF-4Ͱ with affinities that were not significantly affected by the chain length or by the degree of side chain saturation.

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