By Alfred Jost, Solange Magre (auth.), José M. Cuezva, Ana M. Pascual-Leone, Mulchand S. Patel (eds.)
The annual assembly of the Perinatal Biochemical team of the Spanish Biochemical Society held in Madrid on December 15-16, 1989, supplied an outstanding chance to assemble a gaggle of exclusive investigators either from Spain and from in another country with a typical curiosity in developmental endocrinology and biochemistry. the purpose of the symposium was once to give and talk about the newest advancements within the components of endocrine and biochemical procedures serious to basic development and maturation of the baby. to accomplish a excessive measure of interplay one of the members, topic studies in addition to brief communications have been contain within the application. The experiences supplied in-depth details on chosen vital issues. the aim of brief communications was once fold: (i) to supply a discussion board to debate on-going investigations on similar components; and (ii) to provide possibilities for energetic participation through younger investigators. This structure proved very winning in producing fruitful discussions one of the members. Taken jointly the evaluate chapters and the fast communications have led to a coherent and unified topic presentation. Advances in biochemistry, molecular biology and telephone biology have supplied not just new and interesting experimental ways but additionally have unfolded new instructions within the research of differentiation and developmental techniques at mobile, molecular and biochemical degrees throughout the early levels of progress and maturation. in recent times a wealth of knowledge in those parts of improvement has emerged at a swift rate.
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Additional resources for Endocrine and Biochemical Development of the Fetus and Neonate
These observations therefore suggest that the deficient insulin response to 40 glucose in the fetal B-cell reflects an immature glucose oxidation leading to an impaired regulation of the otherwise normal G-channel. In summary the immature B-cell in the fetal pancreas displays an impaired responsiveness to glucose despite relatively adequate insulin reserves and insulin biosynthetic capacity: such poor recognition of the secretory signal seems to be specific of the glucose molecule since a variety of other secretagogues were able to elicit insulin secretion.
We have already discussed more extensively the possibility that they are (21). Autonomy of the pituitary-thyroid feedback system in human fetuses during the third trimester of pregnancy is well established, but the finding that fetal thyroid failure is accompanied by high TSH levels at birth does not exclude that TSH would have been higher (and cord-blood T4 lower) if there had been no transfer of hormones from the mother to the hypothyroid fetus. Transfer of maternal thyroid hormones to athyreotic fetuses would explain that in some of them T4 levels are clearly detectable in cord-blood, and then decrease rapidly (50).
Res 19:90 (1987). Morell, Renin-angiotensin system in hypothyroid rats: effects of potassium iodide and triiodothyronine, Acta Endocrinologica (Kbh) 105:505 (1984). Res 13:691 (1981). 31 DEVELOPMENT OF THE PANCREATIC B-CELLS: GROWTH PATTERN AND FUNCTIONAL MATURATION Bernard Portha Laboratoire de Physiologie du Developpement-GR Regulations Metaboliques et Diabete-CNRS UA-307 Universite Paris 7, Tour 33 75252 Paris Cedex 05, France I NTRODUCTI ON The pancreatic B-cell may be regarded as a biological sensor which plays a key role in controlling the disposal of glucose.