By Gisela Dallenbach-Hellweg, Magnus Knebel Doeberitz, Marcus J. Trunk

The hot version of this atlas integrates all major advances made long ago 15 yearsin molecular pathology, tumor virology, and genetics of cervical melanoma. It emphasizesthe significance of those advances in facilitating its pathological analysis and in optimizingclinical administration and prognosis.A new bankruptcy on immunohistochemistryhas been additional, which include sophisticated detection equipment, e.g., the overexpression ofp16INK4a as a molecular marker within the early differential analysis of premalignant lesions.The part on etiology and pathogenesis in human papillomavirus-inducedneoplasia has been included to symbolize new insights into the sequences of cellularand nuclear deregulation on the molecular level.All chapters were revised to includethe most recent advances and proper stories in easy methods to interpret and managecervical affliction; they're supported by way of the addition of 35 new microphotographic illustrations.The tumor nomenclature is tailored to the most recent version of the WHO classification;the morphology code of the foreign category of illnesses for oncology(ICD-O) has been added.We have additionally up to date the checklist of references via including recentrelevant publications.Again, the workers of Springer-Verlag deserve our thank you for his or her persistence and talent inpreparing the manuscript and in reproducing the microphotographs.

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Additional resources for Color atlas of histopathology of the cervix uteri.

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H&E 25 26 Normal Histology, Regeneration, and Repair Fig. 22. Hyperplasia of reserve cells differentiating into squamous metaplasia. Immunohistochemical reaction with cytokeratin 13 Fig. 23. Monocellular mucin formation in squamous metaplasia. H&E Descending Repair Fig. 24. Monocellular and multicystic mucin formation in squamous metaplasia. H&E Fig. 25. Squamocolumnar junction with original squamous epithelium and adjacent reserve cell hyperplasia underneath the columnar epithelium. Immunohistochemical reaction with cytokeratin KA 1 (from Franke et al.

67) or smooth (Fig. 68). When these polyps protrude through the external os, the surface epithelium may be replaced by reserve cell hyperplasia (Fig. 68), which can differentiate to squamous metaplasia and finally to mature stratified squamous epithelium. When such polyps are completely overgrown by squamous epithelium, they are then classified as ectocervical polyps (Fig. 69).

Glandular (adenomatous) hyperplasia of endocervix with pseudostratified nuclei. H&E Fig. 63. Microglandular hyperplasia of endocervix. H&E Effects of Exogenous Gestagens Fig. 64. Microglandular hyperplasia of endocervix, with reserve cell hyperplasia. H&E Fig. 65. Microglandular hyperplasia of endocervix. H&E, higher magnification 53 54 Hormonally Induced Changes Glandular Papillary Ectropion (Fig. 66) As a result of excessive proliferation and growth pressure, the endocervical mucosa frequently protrudes onto the ectocervical surface (Fig.

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