By A. Bulikova
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Extra info for Antiphospholipid Syndrome
Humangenetik. 5(4), 291-293. Henry ML, Everson B & Ratnoff OD. (1988) Inhibition of the activation of Hageman factor (factor XII) by beta 2-glycoprotein I. J Lab Clin Med. 111(5), 519-523. Hunt JE, Simpson RJ & Krilis SA. (1993) Identification of a region of beta 2-glycoprotein I critical for lipid binding and anti-cardiolipin antibody cofactor activity. Proc Natl Acad Sci U S A. 90(6), 2141-2145. Ikematsu W, Luan FL, La Rosa L, Beltrami B, Nicoletti F, Buyon JP, Meroni PL, Balestrieri G & Casali P.
They found that significant differences were limited to the HLA class II region. In fact, DR4 and DRw53 were found with increased frequency in patients compared with controls, whereas DR3 was absent in all patients. No significant associations between any DQB alleles or C4 or 21-hydroxylase gene polymorphisms and primary APS were found, and the prevalence of DQw7 was not significantly increased in patients. , 2001). The most striking association was found between DQB1*0604/5/6/7/9-DQA1*0102-DRB1*1302 and the presence of antib2GPI antibodies in primary APS.
A. In vivo modulation of angiogenesis by beta 2 glycoprotein I. J Autoimmun 35:232-240. , and Cohen, H. 2003. Paradoxical association between the 316 Trp to Ser beta 2-glycoprotein I (Beta2GPI) polymorphism and anti-Beta2GPI antibodies. Br J Haematol 120:529-531. , and Thiagarajan, P. 2000. Primary antiphospholipid antibody syndrome with mutations in the phospholipid binding domain of beta(2)glycoprotein I. Am J Hematol 65:160-165. 2 Structural Changes in β2-Glycoprotein I and the Antiphospholipid Syndrome Çetin Ağar1,2, Philip G.