By Stephen Chapman, Richard Nakielny

This renowned source assists readers in honing their wisdom of radiological differential analysis for the main normally encountered stipulations in all parts of the physique. the 1st part provides lists of differential diagnoses, supplemented via notes on priceless proof and discriminating elements. the second one part deals unique descriptions at the attribute radiological visual appeal of greater than 80 person ailments. the result's a handy research instrument for examination instruction, in addition to a valuable fast reference for medical practice.Features a very redesigned format, making info much more accessible.Includes up-to-date content material throughout-including the entire diversity of more moderen imaging modalities.Offers absolutely revised content material that displays the most recent in scientific practiceWith six extra contributing specialists.

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M c A l i s t e r W . H . Radiol. Clin. , (1991) 29(1): Inherited diseases in b o n e density in children. 149-64. 33 OSTEOMALACIA AND RICKETS* VITAMIN D DEFICIENCY 1 . Dietary. 2 . Malabsorption. RENAL DISEASE 1. Glomerular disease (renal osteodystrophy*). 2 . Tubular disease (a) Renal tubular acidosis (i) Primary — sporadic or hereditary. (ii) Secondary. g. cystinosis, galactosaemia, Wilson's disease, tyrosinosis, hereditary fructose intolerance. g. lead, cadmium, beryllium. g. amphotericin B, lithium salts, outdated tetracycline, ifosfamide.

Rhabdomyosarcoma. 6. Retinoblastoma. 7. Ewing's sarcoma — lung metastases much more common. 8. Osteosarcoma* — lung metastases much more common. R. ( 1 9 9 7 ) L e u k a e m i a a n d l y m p h o m a in c h i l d h o o d . 1495-516. Radiol. Clin. D. E. (1981) s o l i t a r y b o n e lesions. Radiol. Clin. , R a d i o l o g i c a n d p a t h o l o g i c analysis of 19: 7 1 5 - 4 8 . 22 LUCENT BONE LESION IN THE MEDULLA WELL-DEFINED, MARGINAL SCLEROSIS, NO EXPANSION Indicates a slowly progressing lesion.

Soft-tissue calcification. Dense metaphyseal bands and widened zone of provisional calcification. BONES 11 4. Chronic hypervitaminosis A — not before 1 year of age. Failure to thrive, hepatosplenomegaly, jaundice, alopecia and haemoptysis. Cortical thickening of long and tubular bones, especially in the feet. Subperiosteal new bone. Normal epiphyses and reduced metaphyseal density. The mandible is not affected (cf. Caffey's disease). IDIOPATHIC 1. 14. 2. Idiopathic hypercalcaemia of infancy — probably a manifestation of hypervitaminosis D.

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